Few things are better than LYMErix/OspA for activating Epstein-Barr

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Just adding to the Not-Really-News that Lyme is a form of post-sepsis syndrome with reactivated herpes viruses and tolerance to other TLR-agonist types,…  proving that ILADS commits malpractice, that the ALDF.com criminally hid the fact that OspA injections caused a chronic Lyme-like illness,…  and that that was the reason these ALDF criminals (CDC, Yale, NYMU, UConn cabal) deleted the chronic neurologic (post-sepsis-like outcome) outcome of Lyme from the case definition at Dearborn in 1994.
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We have ABSOLUTE PROOF that the ALDF.com cabal knew OspA was causing a chronic neuro Lyme like illness BEFORE the “case definition” was changed at Dearborn and before Steere committed the research fraud that introduced the ELISA which excludes all but HLA-linked hypersensitivity (bad knee only) Lyme in that first step.  So, there was no “mistake” here, when they raised the cutoff to exclude post-sepsis Lyme.  In fact it may have been to SPECIFICALLY also assure no one was treated with antibiotics for this viral condition that Steere thoroughly discussed with Paul Duray in 1988:
http://www.actionlyme.org/clinical-pathologic-correlations-of-lyme-disease-by-stage-Steere-Duray.pdf
Their slander and libel is the critical element needed to show deliberate “intent to cause harm.”  That’s a very serious criminal charge and not “negligence.”

[See more about this Steere/Duray report from 1988 below and as far as “stages.”  If it takes only 3-7 days for peak proliferation/activation of Epstein-Barr after co-exposure with other TLR agonists including OspA or the Osps, then we know the “stages” of Lyme are really about the “stages” of the post-sepsis like outcome of Lyme.  It’s not really about spirochetes after the first week or so.]

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When is Medical Malpractice a Crime? | Nelson MacNeil Rayfield

 

 

 

And when the ALDF.com decided to whack people with LYMErix knowing the harm it caused by 1993, that’s technically an “assault” charge.  There is more to this…
https://injury.findlaw.com/accident-injury-law/recklessness.html
See http://www.actionlyme.org/UN_PETITION.htm

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OspA was never a vaccine, the main thing responsible for activating EBV is in fact, the fungal Osps, and ILADS still does not explain how any of their alleged treatments address the tolerance and cross tolerance caused by these fungal antigens (the mechanism of illness).  They in no way EVER showed how Lyme caused illness and deliberately neglected the fact that OspA alone was causing the same immunosuppression-AIDS like disease.
Normally “medicine” is about identifying a mechanism of illness and discovering some means to block that mechanism, but in the case of tolerance, it cant be reversed by any means we know of.  ILADS.org never mentions anything about anything.  Go ahead and see for yourselves.

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Toll-Like Receptor Agonists Synergistically Increase Proliferation and Activation of B Cells by Epstein-Barr Virus 

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2838115/

 

181106_SUPPRESSIVE_OSPA

 

A Lipoylated Metabolic Protein Released by Staphylococcus aureus Suppresses Macrophage Activation.

“We demonstrate that lipoyl-E2-PDH is also released by S. aureus and moonlights as a macrophage immunosuppressant by reducing Toll-like receptor 1/2 (TLR1/2) activation by bacterial lipopeptides. A LipA-deficient strain induces heightened pro-inflammatory cytokine production, which is diminished in the absence of TLR2. During murine systemic infection, LipA suppresses pro-inflammatory macrophage activation, rendering these cells inefficient at controlling infection. These observations suggest that bacterial metabolism and immune evasion are linked by virtue of this moonlighting protein.”
https://www.ncbi.nlm.nih.gov/pubmed/29056428

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See more at:  https://www.ncbi.nlm.nih.gov/pubmed/?term=S.+aureus+and+TLR2+and+TLR1

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CHECK THIS OUT and COMPARE IT TO HOW SOON AFTER CO-EXPOSURE with TLR agonists, does EBV reactivate:

http://www.actionlyme.org/clinical-pathologic-correlations-of-lyme-disease-by-stage-Steere-Duray.pdf

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181106_STEERE_DURAY_1

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181106_STEERE_DURAY_2

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181106_STEERE_DURAY_3

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And now from the first report above where they talk about how fast EBV reactivates in the presence of toll like receptor agonists:
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2838115/

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3 to 7 days.

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