Adverse Effects of Vaccines: Evidence and Causality.
Adverse Effects of Vaccines: Evidence and Causality.
Committee to Review Adverse Effects of Vaccines; Institute of Medicine; Stratton K, Ford A, Rusch E, et al., editors.
Washington (DC): National Academies Press (US); 2011 Aug 25.
”The committee identified 18 publications reporting encephalitis or meningoencephalitis after the administration of vaccines containing measles, mumps, and rubella alone or in combination. Mustafa et al. (1993) described one case of encephalitis developing after administration of a MMR vaccine; however, wild-type measles virus was demonstrated in the patient. Fourteen publications did not provide evidence beyond temporality (Ehrengut and Zastrow, 1989; Fescharek et al., 1990; Forster and Urbanek, 1982; Jagdis et al., 1975; Jorch et al., 1984; Kumar et al., 1982; Landrigan and Witte, 1973; Pollock and Morris, 1983; Ross and Yeager, 1977; Schneck, 1968; Schuil et al., 1998; Shuper, 2011; Wiersbitzky et al., 1992b, 1993a). In addition, five publications reported concomitant infections that could contribute to the development of symptoms (Ehrengut and Zastrow, 1989; Forster and Urbanek, 1982; Jorch et al., 1984; Wiersbitzky et al., 1992b, 1993a). These publications did not contribute to the weight of mechanistic evidence.”
“Described below are three publications reporting clinical, diagnostic, or experimental evidence that contributed to the weight of mechanistic evidence.
“Bakshi et al. (1996) described a 16-month-old boy presenting with a focal seizure on the right side and left hemipareses and a left gaze preference 5 months after receiving a measles, mumps, and rubella vaccine and 3 days after undergoing bone marrow transplantation. The patient was administered the vaccine prior to being diagnosed with sickle cell trait and a severe combined immunodeficiency. Serum and CSF were negative for bacteria and fungi. Mumps virus was demonstrated in the urine, serum, and CSF. The patient was diagnosed with meningoencephalitis and died 2 months after the onset of symptoms. Pathological examination of the leptomeninges showed chronic and focally prominent meningitis.
“Lacroix et al. (1995) describe a 5-year-old acquired immune deficiency syndrome (AIDS) patient presenting with fever, generalized seizures, and the inability to stand or walk approximately 2 years after vaccination against measles. The patient died months after presenting with neurological symptoms. Retrospective serum analysis showed measles antibody prior to vaccination. Viral cultures of brain samples were negative for measles virus. Frozen sections of basal ganglia, frontal cortex, and white matter were stained with antibodies against measles virus indicating the presence of measles virus in the brain.
Valmari et al. (1987) described a 7-year-old girl presenting with vomiting, headache, twitching of upper extremities, followed by coma lasting for several hours 54 days after receiving a measles, mumps, and rubella vaccine containing the Moraten measles strain and 5.5 years after receiving a measles vaccine containing the Schwarz measles strain. On the day the measles, mumps, and rubella vaccine was administered the patient complained of back pains leading to a diagnosis of acute lymphoblastic leukemia 23 days after vaccination. The patient presented with the symptoms described above 1 day after the fourth methotrexate treatment. Treatment with acyclovir was started and the patient seemed to improve. Measles virus was demonstrated in the CSF. The patient experienced a recrudescence of the neurological symptoms 58 days postvaccination and fever, photophobia, conjunctival inflammation, and a maculopapular rash 63 days postvaccination. Measles virus was demonstrated in the CSF again.
Weight of Mechanistic Evidence
“Encephalitis is considered a complication of infection with wild-type measles, mumps, and rubella viruses (Gershon, 2010a,b; Litman and Baum, 2010). Encephalitis develops in 1:1,000 to 1:2,000 patients infected with measles virus (Gershon, 2010a). In addition many patients upon recovering suffer from neurologic sequelae (Gershon, 2010a). Encephalitis develops in 1:400 to 1:6,000 patients infected with mumps virus (Litman and Baum, 2010). In patients developing early-onset encephalitis upon infection with mumps virus, the damage to the neurons is by direct viral invasion (Litman and Baum, 2010). In patients infected with rubella virus, encephalitis develops in 1:5,000 patients (Gershon, 2010b). The committee considers the effects of natural infection one type of mechanistic evidence.
“The three publications described above, when considered together, did not present evidence sufficient for the committee to conclude the vaccine may be a contributing cause of encephalitis after administration of a measles or MMR vaccine. The patients described in the cases above had demonstrated immunodeficiencies. The publications presented evidence of the detection of viral antigens on frozen sections or the isolation of mumps or measles virus from the patients. However, the authors did not identify the virus as vaccine strain.
”The latency between vaccination and the development of encephalitis in the publications described above ranged from 5 months to 2 years, suggesting persistent viral infection as the mechanism. Direct viral infection and viral reactivation may contribute to encephalitis; however, the publications did not provide evidence linking these mechanisms to MMR vaccine.
“The committee assesses the mechanistic evidence regarding an association between MMR vaccine and encephalitis as weak based on knowledge about the natural infection and three cases.
As you have just seen, it is precisely as we proposed. The kids being damaged from vaccines were immunosuppressed (or got a contaminated vaccine); there is a warning in the MMR about not vaccinating immunosuppressed children; they are throwing out the vaccine failure cases by claiming the reversion to wild type can’t be distinguished from natural infection; and no doctor is given a tool for assessing immune status prior to vaccination.
These pediatric vaccines are One Size Fits All and the CDC says these “adverse events” are “a calculated risk.” Right now the CDC calculates that the risk of 1:60 kids becoming brain damaged for life is a good risk J The CDC/BigPharma make the claim that vaccines are safe – excluding the phrase “for children who are not already sick or immunosuppressed” – and they BLATANTLY claim that this is not a contributing mechanism. We have shown the mechanism of immunosuppression-reactivates-viruses in parallel with the LYMErix and Lyme, and CFIDS/ME post-sepsis syndrome.
Remember from Paul Auwaerter (and there are other reports on “reversion to wild type” in vaccine failure): ”… human tissue-passaged vaccine isolates are highly related to parent vaccine strains (1, 15).
“…However, fatal infections have been documented in immunodeficient children vaccinated with these strains (1, 12, 14, 15). The symptoms of infection occur many months after immunization, and the viruses isolated are similar to the original LA vaccine (1, 15), suggesting that in the absence of an effective host immune response, persistent infection with the vaccine strain can lead to fatal disease. Viruses isolated from these children could potentially represent virulent revertants of the original LA vaccine.”
The following report refutes the entire concept that live, attenuated viral vaccines is preventing disease; one wonders in Lyme, and Chronic Fatigue Syndrome these childhood vaccine disease or naturally acquired infections are not reactivated, too, with the herpes:
“Subclinical [means no spots or lumps or immunosuppression-ish] Infection is Not Uncommon.”
Ugeskr Laeger. 1992 Jul 13;154(29):2008-13.
[Duration of immunity and occurrence of secondary vaccine failure following vaccination against measles, mumps and rubella].
[Article in Danish]
“… In rare cases, rubella re-infection has resulted in infection in utero, so that a slight risk of congenital rubella cannot be entirely excluded after successful vaccination. No extensive systematic investigations of the effect of revaccination have been carried out and, similarly, the optimal interval between two or more vaccinations has not been illustrated in more detail in the literature. Subclinical infection is not uncommon after all three vaccines. Where measles is concerned, immunity may possibly be regarded as a continuum which, depending upon the antibody level, protects the individual from various degrees of clinical disease. If wild virus can be spread via individuals with subclinical infections, it is doubtful whether population immunity (herd immunity), which is necessary to eliminate the three diseases, can be attained in large populations.”
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