OVERVIEW of Cryme and Disease

* The testing for Lyme disease was falsified to pass off bogus vaccines and test kits (Lyme is fungal or is an immunosuppression outcome like chronic Tb) by a cabal called the ALDF dot com originally based at NYMC.  (Now the ALDF has a mailbox in Lyme, CT, where Edward McSweegan has a second home.)  Many ALDF members are CDC staff and patent holders.  ALDF was the fake “non-profit” / advertising firm for these fake tick-borne disease (TBDs) patented products.

* The disease is caused by the spirochetes shedding exosomes that have Osp-ish (triacyl or fungal or TLR2/1-managed) antigens on them, and contain DNA.   TLR2/1 agonists are fungal endotoxins that are more ferocious than the typical lipopolysaccharides (LPS, TLR4-agonists) of typical bacteria (Spirochaeta is an ancient phylum – they’re not regular bacteria).

TLR2/1 agonists are so toxic that the body shuts down the immune system altogether in response to them, because otherwise the human mammal would die from the septic cytokine storm (Medvedev, Harding, Radolf, Redmond, etc.).  The words for this phenomenon are post-sepsis, endotoxin tolerance, immunosenescence, immunoparalysis, acquired or adaptive immune deficiency, etc.

Anthony Fauci owns a patent for the treatment of this early, toxic-state, septic-state, fungal-acquired immune deficiency.

OspA disease or Lyme disease causes chronic inflammation in the brain but with humoral (in the body or blood stream) immunosuppression with low-to-no- antibodies, according to the NIH (Martin, Marques), Diego Cadavid, and Tulane Primate Center (Philipp).

Allen Steere published in 1993 that people without the HLAs for a hypersensitivity response (autoimmune) to fungal antigens or heat shock proteins will not test positive, after publishing in 1986 that you only need band 41 to diagnose Lyme, and in 1990 about “Chronic Neurologic Lyme” (snippet and link, below).

The same year, 1993, Allen Steere falsified the case definition, adding the ELISA, raising the bar on diagnostics such as to exclude all neurologic cases (snippet and link, below).  The chronic, long term neurologic cases of Lyme disease are actually more due to the reactivated latent herpesviruses (Great Imitator causing also ME, Lupus, etc, which are due to EBV, et al) and tolerance to other infections (mycoplasma cause chronic fatigue by hijacking cell metabolism, depolarizing mitochondrial and red blood cell membranes, and preventing oxygen from crossing the red blood cell membrane), which is the very definition of post-sepsis syndrome.

So, “post-Lyme syndrome” is really “post-sepsis syndrome” and that is why antibiotics don’t cure it.  Chronic Fatigue Syndrome or ME or both (or whatever they are calling it) and Fibromyalgia are the same thing: post-sepsis syndrome.  That is why the NIH pays it no attention.  They know what it is, and since everyone already has EBV and CMV etc., they don’t care – it’s not a new infectious disease.  It’s ones everyone already has.

See more examples of the same phenomenon here.


People wonder why Senator Richard Blumenthal, after suing these CDC, Yale and IDSA crooks for Anti-Trust as CT Attorney General, does nothing for us now that he is in Washington.  Well, one could guess that he was told post-sepsis syndrome is incurable. 

Or, he was told that it was an accidental release.  After all, the OspA-B plasmid seems to have been phage-vectored either from mycoplasma or Brucella.


, Lyme borrelia were perceived by the Centers of Disease Control and Prevention (CDC) to be just another group of relapsing fever spirochetes. The CDC formally claimed that the way to test for them was to look for new and expanding IgM (IgM means new antibodies or “new infection,” IgG means “not a new infection”) antibodies by serial, or sequential or follow-up Western Blotting. The nature of relapsing fever’s relapse is antigenic variation. That is, as soon as the mammalian host makes enough antibodies to fight off the spirochetes, a new class of variants would appear, rendering the first set of antibodies useless, and then you have a relapse or a recurrence of the fever and flu-like illness.
That was the model, but was only what they observed. The ones who did not have periodic relapses were probably not recognized as having relapsing fever.   Remember it was only 100 years ago or so that scientists studying syphilis and African diseases became keen enough, technically, to report about what they saw under a microscope.


Let’s take a detour – the old days; nostalgia


PRELIMINARY NOTE: At the first meeting of the Tropical Medicine Section of the British Medical Association in London last year I advanced the view that, in all probability, what might be called the ” infective granule ” would yet be found to play an important part in certain protozoal infections, and more especially in spirochaetosis and trypanosomiasis.  I based this belief on the work of Leishman as regards the changes undergone by Spirochaeta duttoni in Ornithodorus moubata, and on the allied changes which I had found to occur in the Sudan fowl spirochaete when ingested by Arga pericus.  I have been continuing the work on fowl spirochaetosis and have recently arrived at some most interesting and significant results, which may yet have
considerable bearing on the view we must take of the pathology of this and other spirochaetal diseases, and possibly also on their treatment. …
…It will perhaps be remembered that one found intracorpuscular forms in this fowl spirochaetosis, and that following Sambon, one had to come to the conclusion that these endoglobular bodies represented a stage in the lifecycle of the spirochaete — constituted, in short, its stage of schizogony in the fowl.  Sambon, however, who expressed this view from the study of a few slides I gave him, did not indicate how this red cell invasion occurred.  For a long time I believed the spirochaetes themselves entered the red cells and broke up, or coiled up, within them to form these remarkable bodies.  As the parasites can and do enter and leave the erythroblasts of the fowl, there was good ground for this supposition.  Now however, I know better.
By the use of the dark-field method, and more especially by practising liver puncture on chicks at the crisis or on chicks which have been given a sufficiently large dose of salvarsan, I have found that in the liver in particular, also in the spleen and lung, the spirochaetes undergo an astonishing change.  They discharge from their periplastic sheaths spherical granules, and it is apparently these granules which enter the red cells, develop in them and complete a cycle of schizogony. The appearance is very remarkable.  If a well-infected chick be given a dose of salvarsan, the peripheral blood is soon cleared , or nearly cleared of spirochaetes.  If then a drop of liver juice be examined by the dark-field method, it will be found swarming with spirochaetes and with highly refractile granules.  The source of the latter is soon apparent, for attention will be directed to spirochaetes which are not moving in the usual way, but are in a state of violent contortion, or are, so to speak, shaking themselves to and fro.  Indeed, I cannot give a more apt comparison than by likening their movements to those of dogs which have been in water and are shaking themselves vigorously to dry their coats.  The object of the spirochaetes, however, is to rid themselves of the bright sphereical granules which can  be seen within them and which may or may not be aggregations of the so-called chromatin core.  They are forced along the periplastic sheath and suddenly discharged , so that they become free in the medium and dance hither and thither as tiny, solid, spherical, brilliant white particles.  In process of time the spirochaete loses its activity, becomes difficult to see, and eventually all  that is left of it is the limp and lifeless sheath drifting aimlessly in the fluid and liable to be caught up and swept away by some still vigorous parasite. Such a sheath may still retain one or two of the granules which it has been unable to discharge.
As may be imagined, the process is most fascinating to watch, and my observations have been confirmed by Captain Fry and Mr. Buchanan, of these laboratories and Captain O’Farrell, R.A.M.C.   I may also say that the first-named had previously seen a shedding off of granules by trypanosomes in the peripheral blood of experimental animals, a phenomenon which he is now studying.
It is these spirochaete granules in the liver, spleen and lung, and possibly also in other internal organs, which I believe, invade the red cells.  I think I have seen the penetration occur, but require to make further observations in order to be certain as to the mode of entry.  Such a chain of events fully explains all the puzzling features which this intracorpuscular infection has hitherto presented, and moreover, brings it into line with the infective granules found in the ticks, for these very closely resemble those seen in liver-juice films both when examined by dark-field method and when stained by the Levaditi process.  There are various other points more especially as regards the peculiar staining reactions of these granules, into which I need not enter beyond saying that the fact that, when free, they do not appear to take on the Romanowsky stain may explain why they have not previously been noticed.  The work is also not yet complete as it is necessary to find out if the spirochaetes ingested by ticks behave in a similar manner and thereby produce the granules of Leishman.
I see that Jowett in South Africa has recently discovered what appears to be an identical form of fowl spirochaetosis, and I trust he will employ  the dark-field method and endeavour by liver puncture and the use of salvarsan, for the purpose of creating an articficial crisis to follow out the curious cycle I have indicated
From these observations and others which will be fully detailed at a later date I have come to the conclusion that this fowl spirochaete must be classed as a specific entity and I am proposing for it the name Spirochaeta granulosa penetrans, which, though lengthy, suitably indicates its more important peculiarities.  At the same time it is quite possible–nay, even probable –that other pathogenic spirochaetes behave in a  similar manner.  I have found these granules to be resistant forms and their presence in countless numbers in the tissues might explain part of the mechanism of relapse and the difficulty of curing completely some of the more chronic spirochaetal infections, as, for example, syphilis and yaws.
    In conclusion, I must thank Professor Erlich for most kindly placing at my disposal an ample supply of his new and valuable remedy.


How to diagnose relapsing fever borreliosis (in the old days):

Allen Steere published in 1986 that new IgM bands appearing after treatment meant that antibiotics did not kill all the spirochetes. Additionally in the final paragraph of his report that became the basis of the 1990 CDC standard to diagnose Lyme, Steere says it is fine to use only band 41 to diagnose Lyme, even early Lyme:


”… of spirochetes on the solid phase of an ELISA, improved the-sensitivity ofthe test (36). This observation suggests that the 41-kD antigen of B. burgdorferi may give better results in an ELISA for early Lyme disease than the current test, which uses sonicated whole spirochetes (1 1, 12). Furthermore, in this study, IgM antibodies to the 41-kD polypeptide were usually apparent by immunoblots before IgM titers were elevated by the current ELISA (12). Although antibodies reactive against this antigen may be present in patients with relapsing fever or syphilis (1 1, 14), these diseases can be distinguished clinically from Lyme disease and therefore should not cause diagnostic confusion. The binding of this antigen by IgM from rheumatic disease controls was typically very weak and could potentially be blocked to avoid false-positive results in this group of patients.”

Antigens of Borrelia burgdorferi recognized during Lyme disease. Appearance of a new immunoglobulin M response and expansion of the immunoglobulin G response late in the illness.   https://www.ncbi.nlm.nih.gov/pubmed/3531237



Here is the CDC’s 1990 diagnostic standard (no ELISAs), which is based on the Steere, 1986 report above:


The CDC publication says:

“Laboratory criteria for diagnosis

”* Significant change in IgM or IgG antibody response to B. burgdorferi in paired and acute- and convalescent-phase serum samples.”

It says to look for changing and expanding IgM and later IgG antibodies over time with Western Blotting, because relapsing fever spirochetes constantly change their surface proteins and hence, you would see changing antibodies in human or mammalian blood. It is what they do. It is their nature.

Borreliae (the whole genus) undergo constant antigenic variation. The plasmid DNA that encodes the surface ligands called Outer Surface Proteins (Osps) or Variable Major Proteins (Vmps) reassemble, producing new antigen all the time, which is said to be the mechanism of the relapse, in Relapsing Fever. This fact of all Borrelia makes vaccines and valid testing impossible except for a flagellin method for detection and was known by all involved in this scandal, as you have seen, all along, and certainly since 1986.

Yale has a patent for a refined anti-flagellar antibody test, such that the antibodies against the fragment of Borrelia burgdorferi they patented, did not cross react with flagellar antigen from other Borrelia or other flagellated organisms (US Patent 5,618,533). This flagellin test developed in 1990-1991 by Yale employees was “specific” to Borrelia burgdorferi flagellin. “Specificity” is a criterion in the Food and Drug Administration’s (FDA’s) guidelines for the validations of analytical methods, as we will later see.


The ALDF cabal

In the late 1980s and early 1990s, it was decided by mostly the CDC officers in this cabal that they should commercialize Lyme Borrelia and other emerging, tick-borne diseases (TBDs) by patenting vaccines and test kits based on recombinant antigens of this varying type other than flagellin, anyway.  No one knows who gave the CDC the authority to do this, but this decision coincided with the establishment of the fake non-profit, the American Lyme Disease Foundation (ALDF.com), Valhalla, NY, in 1990, by Edward McSweegan, Durland Fish, Gary Wormser, and John J. Connolly [the then-president of New York Medical College in association with Kaiser-Permanente (KP)]. KP is still at NYMC writing MD-training modules.  CDC is often found in collaboration with KP; we knew this even before their “Morgellon’s investigation” scam.


Detour – So many CDC scandals

No one knows exactly why the CDC is involved in so many scandals, all of which make it appear that it is the CDC’s job 1) not to detect diseases on behalf of insurance companies, but 2) only promote and sell vaccines on behalf of pharmaceutical companies or their own personal patents. There does not appear to be any cognitive cohesion in their policies and activities. It’s not just in Lyme disease, does the CDC appear to say, “There are no diseases; there are only vaccines.” The National Institutes of Health have said publicly that approximately 4 million people in the United States have Chronic Fatigue Syndrome and 8 million have Fibromyalgia. If we were to ignore Lyme disease, that is still 12 million people with a disabling, fatiguing illness – more than any other disease or illness – , but the CDC ignores it, when they’re not downright committing research fraud (Suzanne Vernon and her mycoplasma in Chronic Fatigue Syndrome “report”) as regards it.

The very latest CDC scandal at the time of this writing from the CDC is as regards “SPIDER,” or
”A group calling itself CDC Scientists Preserving Integrity, Diligence and Ethics in Research, or CDC SPIDER, put a list of complaints in writing in a letter to the CDC Chief of Staff and provided a copy of the letter to the public watchdog organization U.S. Right to Know (USRTK). The members of the group have elected to file the complaint anonymously for fear of retribution.

“’It appears that our mission is being influenced and shaped by outside parties and rogue interests… and Congressional intent for our agency is being circumvented by some of our leaders. What concerns us most, is that it is becoming the norm and not the rare exception,” the letter states. “These questionable and unethical practices threaten to undermine our credibility and reputation as a trusted leader in public health.’”


The ALDF.com is a False Claims-and-Racketeering organization:   The very same individuals who own the patents for vector borne disease DNA and recombinant antigen test kits and vaccines, were the same ones involved in this fake non-profit, were the same ones who later falsified this former “see Lyme as a relapsing fever organism,” and were the same ones involved with the FDA panel to approve the Lyme vaccines (Steere, Schoen, Barbour, Johnson, McSweegan, etc). That’s a “racketeering influenced and corrupt organization (RICO).”   Via their public slander and libel against very sick people, we can add Color of Law charges against this cabal, which means they abused their power as government officials and employees to deny their victims’ civil rights.


Tick Borne Diseases – the is/do or structure/function, or structure/which Toll Like Receptors (TLRs) manage them?

Most vector borne diseases that are not viral are bearers of fungal antigens (TLR2/1 or TLR2/6 agonists; that is, what they are, fungal antigens, or di-, or triacylated lipoproteins, is how they are managed by toll like receptors) and cause immunosuppression. You can find this out for yourselves:

https://www.ncbi.nlm.nih.gov/pubmed/?term=plasmodium+and+TLR2 (Babesia)


https://en.wikipedia.org/wiki/TLR2 :
Protein-ligand interactions[edit]

“TLR2 resides on the plasma membrane where it responds to lipid-containing PAMPs such as lipoteichoic acid and di- and tri-acylated cysteine-containing lipopeptides. It does this by forming dimeric complexes with either TLR 1 or TLR6 on the plasma membrane.[11]TLR2 interactions with malarial glycophosphatidylinositols of Plasmodium falciparum was shown[12] and a detailed structure of TLR–GPI interactions was computationally predicted.[13]



The chronic agonism of TLR2/1 such as by TBDs bearing fungal antigens causes immunosuppression and this is well known. One finds it happening elsewhere such as in mycobacterial and mycoplasma infections.   Another term for this immunosuppression is endotoxin tolerance.

“Endotoxin tolerance protects the host by limiting excessive ‘cytokine storm’ during sepsis, but compromises the ability to counteract infections in septic shock survivors. It reprograms Toll-like receptor (TLR) 4 responses by attenuating the expression of proinflammatory cytokines without suppressing anti-inflammatory and antimicrobial mediators, but the mechanisms of reprogramming remain unclear. “
2016, Medvedev https://www.ncbi.nlm.nih.gov/pubmed/26457672


Endotoxin tolerance and cross tolerance (where the chronic exposure to fungal antigens expands the tolerance to other types of infections or other TLR agonists such a lipopolysaccharide or viral antigens) is the exact definition of post-sepsis syndrome, with reactivated infections of all kinds.

NIH and Washington University in Saint Louis (wustl.edu) published in 2014 that after sepsis or endotoxin tolerance such as from tick bite fungal shock (think of a tick bite as being injected with bathtub scum, because that is exactly what these fungal antigens in TBDs are), dormant viruses such as Epstein-Barr virus (EBV) and Cytomegalovirus (CMV) re-emerge. The same thing happens when people are too immunosuppressed by these “autoimmune diseases” via their immune-suppressing medications such as Humira or Stelara.   They are warned against not becoming too immunosuppressed by being exposed to fungal diseases because of the risk of lymphoma. Well, what is lymphoma causes by? Reactivated herpesviruses. The same thing happens in transplant patients: if they become too immunosuppressed from their anti-rejection medications they risk the reactivation of Epstein-Barr or lymphoma. These facts are well known:

Don’t become too immunosuppressed with immunosuppression medications or you run the risk of reactivated EBV, CMV, etc – related cancer. Don’t vaccinate an immunosuppressed child with live, attenuated viruses, but use fully dead ones because they child mght them become brain damaged from those neurotropic viruses, such as mumps, measles and rubella. Be careful, if you are an astronaut of medical school student, not to become too stressed out, because cortisol reactivates the herpesviruses causing chronic fatigue. For people who become too immunosuppressed from fungal diseases, the current Director of the National Institute of Allergy and Infectious Diseases (NIAID), Anthony Fauci, has proposed using extra Interleuken-2 (IL-2) to boost the immune system (US patent 5,696,079).



That people with chronic fatigue syndrome have a disease like chronic mononucleosis is exactly right, but due to the immunosuppression there won’t be any or many antibodies. You cant use antibody testing to detect these chronic active, post-sepsis infections


So, how can anyone expect to make vaccines out of these fungal-ish TBDS? Well, JUST LIE :D.

And that was the False Claim aspect of this complex crime.  That was what we call “the Dearborn stunt.” These criminals of the ALDF.com, many of whom were patent-owning CDC officers, falsified the case definition at a bogus consensus conference in Dearborn, Michigan in late 1994, such as to narrow the “case definition” of “Lyme disease,” to no longer claim that Lyme was just another relapsing fever Borrelia, but a whole new thing, “Lyme disease” – only an HLA-linked hypersensitivity response or allergy response.

You have just seen the overview.

Lyme used to be known as a relapsing fever spirochete, none of the spirochetal diseases are curable (spirochetes are not eradicable), the CDC is corrupt and everyone knows it, you cant make vaccines out of fungal antigens, the disease is an immunosuppression one (also called post-sepsis or endotoxin tolerance), the ALDF.com was the original RICO organization who falsified the testing for Lyme to suit their own bottom lines, and also slandered and libeled all kinds of sick people which is a denial of civil and human rights by “Color of Law” or by government officials or employees (CDC and NIH staff):



“Section 242 of Title 18 makes it a crime for a person acting under color of any law to willfully deprive a person of a right or privilege protected by the Constitution or laws of the United States.

For the purpose of Section 242, acts under “color of law” include acts not only done by federal, state, or local officials within the their lawful authority, but also acts done beyond the bounds of that official’s lawful authority, if the acts are done while the official is purporting to or pretending to act in the performance of his/her official duties. Persons acting under color of law within the meaning of this statute include police officers, prisons guards and other law enforcement officials, as well as judges, care providers in public health facilities, and others who are acting as public officials. It is not necessary that the crime be motivated by animus toward the race, color, religion, sex, handicap, familial status or national origin of the victim.

“The offense is punishable by a range of imprisonment up to a life term, or the death penalty, depending upon the circumstances of the crime, and the resulting injury, if any.

“TITLE 18, U.S.C., SECTION 242

“Whoever, under color of any law, statute, ordinance, regulation, or custom, willfully subjects any person in any State, Territory, Commonwealth, Possession, or District to the deprivation of any rights, privileges, or immunities secured or protected by the Constitution or laws of the United States, … shall be fined under this title or imprisoned not more than one year, or both; and if bodily injury results from the acts committed in violation of this section or if such acts include the use, attempted use, or threatened use of a dangerous weapon, explosives, or fire, shall be fined under this title or imprisoned not more than ten years, or both; and if death results from the acts committed in violation of this section or if such acts include kidnaping or an attempt to kidnap, aggravated sexual abuse, or an attempt to commit aggravated sexual abuse, or an attempt to kill, shall be fined under this title, or imprisoned for any term of years or for life, or both, or may be sentenced to death.”




Before we begin with a Chronology of the Cryme, let’s focus on 4 OTHER reports by the primary perpetrator of the crime of falsifying the testing, Allen Steere – the one here who said you only needed band 41 or an anti-flagellar antibody to diagnose Lyme, and who knew that there were 2 distinct outcomes to this allegedly new Borreliosis (the one with the OspA-B plasmid), that sometimes conferred an HLA-linked arthritis outcome in addition to the typical chronic neurologic and New Great Imitator-like outcome that the majority suffer:

1) Antigens in Europe, as I call it, which shows how Allen Steere in 1993 falsified the testing by adding the ELISA, and raising the total concentration of antibodies cutoff to exclude neurologic Lyme,

J Infect Dis. 1994 Feb;169(2):313-8.

Antibody responses to the three genomic groups of Borrelia burgdorferi in European Lyme borreliosis.

Dressler F1, Ackermann R, Steere AC.




2) the 1993 Steere report from Europe at the same time that was republished in the Dearborn booklet, where he allegedly prospectively assessed this new proposal, but which is probably research fraud also,

J Infect Dis. 1993 Feb;167(2):392-400.

Western blotting in the serodiagnosis of Lyme disease.

Dressler F1, Whalen JA, Reinhardt BN, Steere AC.


3) the 1993 report where Steere states that antibodies against the fungal Osps are prominent in people with the genetic background for a hypersensitivity response, whereas the people with meningitis or neurologic Lyme don’t,

Infect Immun. 1993 Jul;61(7):2774-9.

Association of treatment-resistant chronic Lyme arthritis with HLA-DR4 and antibody reactivity to OspA and OspB of Borrelia burgdorferi.

Kalish RA1, Leong JM, Steere AC.



“Compared with the 80 patients with arthritis, only 1 of the 5 patients who had chronic neuroborreliosis and who never had arthritis showed weak reactivity to the Osp proteins (P = 0.03)” — Allen Steere.

And that is what WE said 😀

4) Steere’s 1990 report called “Chronic Neurologic Lyme,” where he very clearly recognizes this other, non-arthritis outcome that was later left out of the 1994 “Dearborn case definition.”



And now a bit about the nature of slander and libel:

30 He that is not with me, is against me: and he that gathereth not with me, scattereth.

31 Therefore I say to you: Every sin and blasphemy shall be forgiven men, but the blasphemy of the Spirit shall not be forgiven.

32 And whosoever shall speak a word against the Son of man, it shall be forgiven him: but he that shall speak against the Holy Ghost, it shall not be forgiven him, neither in this world, nor in the world to come.

33 Either make the tree good and its fruit good: or make the tree evil, and its fruit evil. For by the fruit the tree is known.

34 O generation of vipers, how can you speak good things, whereas you are evil? for out of the abundance of the heart the mouth speaketh.

35 A good man out of a good treasure bringeth forth good things: and an evil man out of an evil treasure bringeth forth evil things.

36 But I say unto you, that every idle word that men shall speak, they shall render an account for it in the day of judgment.

37 For by thy words thou shalt be justified, and by thy words thou shalt be condemned.



And now for some examples of the slander and libel

“Telling women and girls inaccurately that they have Lyme disease “condemns patients to long-term, untreated debility and  useless, toxic and expensive courses of antibiotics,” Sigal wrote  in an editorial in the May 15 issue of the journal Hospital   Practice.”–Lenny Sigal (Munchmeister), Emerging diseases // Ticks carry multitude of threats // ( Minneapolis Star Tribune ) Gordon Slovut; Staff Writer; 06-05-1996


“With widespread anxiety about Lyme disease has come Munchausen syndrome and Munchausen syndrome-by-proxy in those concerned about “chronic” Lyme disease.– Leonard ***Sigal***, MD, in Rahn and Evan’s, 1998 book, “Lyme disease” ACP Key Diseases series, Page 149.



“LYME PARANOIA”– Robert Schoen, MD, Yale Rheumatology

“Lyme fear prevails more than disease”, The Washington Times , Dr. Mary Jane Minkin;

“Q: I thought I might have Lyme disease because I had several of the symptoms: joint pain, fatigue and headaches. But my blood test was negative. Could I have Lyme disease anyway?

“A: It’s unlikely, but you’re not the first person to express concern to me about having Lyme disease in spite of a negative blood test.

“While it’s especially important at this time of year to be aware of the warning signs of the disease – a skin rash around the site of a tick bite, headache, fever, fatigue and muscle or joint pain – ***Lyme paranoia,*** as I call it, is not warranted. Even in high-risk areas such as Connecticut, where I live, the chance of developing Lyme disease after a tick bite is only 1 to 2 percent.

“Many patients come in with relatively nonspecific symptoms, believing they have Lyme disease,” says noted Lyme researcher and rheumatologist Dr. Robert Schoen, clinical professor of medicine at Yale University School of Medicine….”


“LYME ANXIETY”– Eugene Shapiro, Yale Pediatrician

“The degree of anxiety about Lyme disease that many people have doesn’t appear to be justified, given the positive long-term treatment outcomes we’ve observed.”



“Without the Lyme disease vaccine, we’re back to ground zero in terms of how we’re going to combat the Lyme disease epidemic,” says Fish, conference program co-chair…Companies “may not want to take on (development of) a vaccine for a disease that is treatable and has a relatively low incidence.”

-Durland Fish, in USA Today quoted by Anita Manning, August 21, 2002

He proceeds down the list, name by name: “Totally bogus.” “He killed one of his patients.” “They tried to shut him down.” Words like “crackpot,” “wacko,” “buffoon” and “fraud” pepper his discourse. A little later, he stops to ponder a question. “I don’t know,” he says after a moment’s thought. “I don’t know why they hate me so much.”

-State Joins Dispute Over Lyme- Scientists Battle Over Treatment.
By Michael Regan| (Hartford, CT) Courant Staff Writer, August 24, 2007

“He had a degree in biology and wanted to do graduate work on mammals. But with the draft looming, he opted instead for an occupational deferment. “I went to work for the state health department, totally bored out of my mind inspecting restaurants. A terrible job, and I wasn’t very good at it,” Fish said. … “I’m not a very good cop. … I used to coach them on how to get by – how to cheat.”

-State Joins Dispute Over Lyme- Scientists Battle Over Treatment.
By Michael Regan| (Hartford, CT) Courant Staff Writer, August 24, 2007


“Lyme is a socially acceptable disease.  You can talk about it any cocktail party.”

– David Weld, head of the ALDF.com in USA Today


More at:


Slander and libel, anyone?  Color of Law Abuses?  Denying civil rights to chronic neurologic/post-Lyme sepsis patients?




























































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